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治疗性S100A8/A9阻塞抑制心肌和全身炎症,并减轻败血症引起的心肌功能障碍

Gabriel Jakobsson1,2, Praveen Papareddy3, Henrik Andersson4

  • 1Department of Translational Medicine, Lund University, Lund, Sweden.

Critical care (London, England)
|September 29, 2023
PubMed
概括

阻断S100A8 / A9,一种促炎蛋白,有效地治疗小鼠的败血症诱导的心肌功能障碍. 这种方法表明,作为严重败血症患者心脏问题的新疗法策略有前途.

关键词:
末端毒性病 (Endotoxemia) 是一种导致内毒性病的疾病.炎症 炎症是一种炎症.线粒体功能 线粒体功能中性粒细胞中性粒细胞.S100A8 / A9 / A9 / S100A8 / A9 / S100A8 / A9 / S100A8 / A9 / S100A9 / S100A8 / S100A9 / S100A9 / S100A8 / S100A9 / S100A9 / S100A9 / S100A8 / S100A9 / S100A9 / S100A9 / S100A8 / S100A9 / S100A9 / S100A8 / S100A9 / S100A9 / S100A8 / S100A9 / S100A9 / S100A8 / S100A9 / S100A8 / S100A9 / S100A9 / S100A8 / S100A9 / S100A8 / S100A9 / S100A8 / S100A9 / S100A8 / S100A9 / S100A8 / S100A8 / S100A9 / S100A8 / S100A9败血症引起的心肌功能障碍

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