通过向NF-κB和AP-1通路,KDM2B调节了败血症的炎症和氧化应激

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Xin Li1, Xinyu Tian1, Dongliang Zhang1

  • 1Department of Nephrology, Beijing Jishuitan Hospital, Capital Medical University, Beijing, China.

Immunity, inflammation and disease
|September 29, 2023
PubMed
概括

氨酸特异性去甲基酶2B (KDM2B) 阻断通过减少炎症和氧化应激来防止败血症引起的急性损伤 (AKI). 这表明KDM2B是治疗毒症相关AKI的潜在治疗标.

关键词:
AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1 AP-1这就是KDM2B.在 NF-κBB 中.细胞的增殖细胞的增殖.这是一种炎症炎症炎症炎症.氧化应激是一种氧化应激.已经有了血症.

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