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Updated: Jul 15, 2025

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白血病突变FLT3-ITD被保留在树突细胞中,并破坏其平衡,导致扩大Th17频率

Patrick A Flynn, Mark D Long, Yoko Kosaka

    bioRxiv : the preprint server for biology
    |October 2, 2023
    PubMed
    概括

    在急性髓性白血病 (AML) 中激活FLT3突变会破坏树突细胞 (DC) 表型,将它们倾向于促进Th17细胞的cDC2子集,影响瘤微环境.

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