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突变的SF3B1促进了PDAC中的恶性瘤.

Patrik Simmler1,2, Eleonora I Ioannidi2, Tamara Mengis2

  • 1Department of Biology, Institute of Molecular Health Sciences, ETH Zurich, Zurich, Switzerland.

eLife
|October 12, 2023
PubMed
概括

SF3B1K700E突变驱动胰腺癌的攻击性,特别是KRAS和p53突变. 这种拼接因子阻碍了TGF-β1的产生.

关键词:
这是一个PDAC鼠标模型.在TGFb信号传输中.灭症 (apoptosis) 是一种死亡的过程.癌症生物学 癌症生物学人类癌细胞是人类的癌细胞.这里是鼠标鼠标鼠标鼠标鼠标鼠标.类PDAC有机物

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