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Updated: Jul 13, 2025

Dual CRISPR-Interference Strategy for Targeting Synthetic Lethal Interactions Between Non-Coding RNAs in Cancer Cells
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一种穿透细胞的质破坏转录因子CP2c复合体诱导癌症特异性的合成死亡率

Seung Han Son1, Min Young Kim1, Sungwoo Choi1

  • 1Department of Life Science and Research Institute for Natural Sciences, College of Natural Sciences, Hanyang University, Seoul, 04763, South Korea.

Advanced science (Weinheim, Baden-Wurttemberg, Germany)
|October 16, 2023
PubMed
概括

一种新,ACP52C,通过破坏特定蛋白质复合体和降解关键蛋白质,触发癌症细胞死亡,为癌症药物开发提供了一条新的途径.

关键词:
CP2c复合物的破坏一种细胞透性.这是一种全方位抗癌药物.合成杀伤性 合成杀伤性转录因子 CP2cc 的转录因子.

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