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ZmELF3.1集成了RA2-TSH4模块,以抑制玉米枝的分枝

Yurong Xie^1,2, Yongping Zhao¹, Lihong Chen³

¹Biotechnology Research Institute, Chinese Academy of Agricultural Sciences, Beijing, 100081, China.

The New phytologist

|October 20, 2023

概括

ZmELF3.1 规范了玉米枝分支编号 (TBN). 功能丧失突变物通过改变ELF3-ELF4-RA2复合体来增加TBN,影响TSH4表达并为遗传改进提供目标.

科学领域:

植物分子生物学植物分子生物学
玉米的遗传学玉米的遗传学
农业学是一种农业学.

背景情况:

塔塞尔分支数 (TBN) 对于玉米适应高密度种植和谷物产量至关重要.
控制TBN的分子机制尚不清楚.
众所周知,ZmELF3.1通过晚上复合体来调节开花时间.

研究的目的:

阐明ZmELF3.1在玉米中的TBN调节中的作用.
确定涉及TBN控制的分子相互作用和途径.

主要方法:

使用了分子和遗传研究.
对ZmELF的功能丧失突变的分析3.1.1.
研究蛋白质与蛋白质相互作用 (ZmELF3.1,RA2,ZmELF4.2) 和DNA结合试验 (RA2到TSH4促进剂).

主要成果:

丧失功能的ZmELF3.1突变体在TBN.显著增加.
发现RA2直接抑制TSH4的表达,减少TBN.
ZmELF3.1与RA2和ZmELF4.2形成三蛋白复合体,增强RA2对TSH4.4的抑制作用.

结论:

ZmELF3.1在调节玉米中的TBN方面发挥着至关重要的作用.
一个涉及ELF3-ELF4-RA2复合体和miR156-SPL模块的新型调节途径控制了TBN.
这项研究为玉米中TBN的遗传改进提供了有价值的目标.

关键词:

在RA2 RA2 RA2 RA2 在TSH4上在 ZmELF3.1中,我们可以看到玉米玉米玉米是一种塔塞尔分支机构的分支机构号码 (TBN)

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