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Updated: Jul 12, 2025

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DMOG通过HIF-1通路在先天性淋巴细胞中保护小鼠IL-33诱导的肺2型炎症

Anupriya Gupta1, Chang Ook Park2, Kwonik Oh3

  • 1Department of Pathology, Hallym University College of Medicine, Chuncheon, South Korea.

Biochemical and biophysical research communications
|October 28, 2023
PubMed
概括

由DMOG模仿的缺氧通过降低IL-5和IL-13在先天性淋巴细胞中的调节来减少2型肺炎. 这表明prolyl氧酶-缺氧-诱导因子通路是肺炎的潜在治疗点.

关键词:
在 DMOG 中,DMOG 是 DMOG.氧气过低是因为缺氧.它们有IL-13和IL-13.在IL-5中,IL-5是最常见的.肺炎是一种肺炎.

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When T cells with CD4 markers are activated, they give rise to two types of effector cells: helper T cells and regulatory T cells. Meanwhile, T cells with CD8 markers differentiate into effector cytotoxic T cells. The differentiation of CD4 T cells into helper T cell subsets, such as Th1, Th2, and Th17 cells, is dependent on the antigen type, antigen-presenting cell, and regulatory cytokines.
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