黑素通过BMAL1/ROS/MAPK-p38途径诱导RAW264.7细胞亡,以改善绝经后的骨质疏松症

Xiaochuan Wang1, Wen Jiang1, Kexin Pan1

  • 1Department of Orthopedics, First Hospital of China Medical University, Shenyang, China.

Bone & joint research
|October 31, 2023
PubMed
概括

黑色素通过BMAL1/ROS/MAPK-p38通路促进巨细胞的亡,从而增强绝经后骨质疏松症患者的骨质量. 这项研究揭示了黑激素.

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