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Updated: Jul 11, 2025

Methods for Evaluating the Role of c-Fos and Dusp1 in Oncogene Dependence
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Published on: January 7, 2019

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鉴定DUSP4/6过度表达作为NRAS诱导的肝癌发生的潜在静态

Sophie Klemm1, Katja Evert1, Kirsten Utpatel1

  • 1Institute of Pathology, University of Regensburg, Regensburg, Germany.

BMC cancer
|November 10, 2023
PubMed
概括

神经母细胞瘤RAS病毒性瘤基因同源 (NRAS) 突变可以在小鼠中启动肝癌. 过度表达的双特异性酸酶4和6 (DUSP4/6) 和CD133可能是肝细胞癌的治疗点.

关键词:
CD133 是一个CD133 CD133 是一个CD133胆性瘤是一种胆性瘤.这就是DUSP4P.肝细胞癌是肝细胞癌.通过MAPK路径.它们是NRAS和NRAS.

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