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Updated: Jun 25, 2026

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通过ac4C介导的转录基因调节,NAT10参与心脏重塑

Jing Shi1, Chuanxi Yang2, Jing Zhang1

  • 1Department of Cardiology (J.S., K.Z., J.Z., P.L., X.W., W.S., X.K.), The First Affiliated Hospital of Nanjing Medical University, Nanjing Medical University, China.

Circulation research
|November 13, 2023
PubMed
概括

在心脏重塑中,NAT10介导的N4-乙基丁 (ac4C) 乙化升高. 抑制NAT10可以减少心脏缩和纤维化,这表明ac4C修饰是心力衰竭的治疗点.

关键词:
通过乙化处理.过度缩的过度缩是一个问题.鼠标 鼠标 鼠标 鼠标

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