在HPA和SAM轴中介于压力下的创造力受损
Xiaoyu Guo1,2, Yifan Wang1, Yuecui Kan3
1Key Laboratory of Modern Teaching Technology, Ministry of Education, Shaanxi Normal University, Xi'an, China.
Psychophysiology
|November 16, 2023
概括
急性压力会通过降低认知灵活性来损害创造力. 通过皮质醇,眼率和瞳孔直径表明的生理兴奋,会对不同的思维和原创性产生负面影响.
科学领域:
- 神经科学是一个神经科学.
- 认知心理学 认知心理学
- 压力研究 压力研究
背景情况:
- 由于压力,个人的创造力受到现代社会环境的挑战.
- 关联急性压力与创造性认知处理的机制尚不清楚.
研究的目的:
- 研究神经内分泌对压力的反应及其对创造力的影响.
- 探索认知灵活性在调解压力对创造力影响中的作用.
主要方法:
- 使用ELISA评估唾液皮质醇 (HPA轴标记物).
- 测量了SAM轴活动的眼 rate (多巴胺指标) 和瞳孔直径 (北上腺素指标).
- 评估认知灵活性 (威斯康星州的卡片排序任务),分歧思维 (替代用途任务) 和融合思维 (远程关联任务).
主要成果:
- 与对照组相比,压力组表现出更高的皮质醇增加和眼率和瞳孔直径的增加.
- 皮质醇升高会损害不同思维的独创性,降低认知灵活性 (增加坚持性错误).
- 眼率和瞳孔直径与错误增加和原创性降低相关.
结论:
- 在急性压力下生理兴奋会损害不同的思维.
- 神经内分泌路径,特别是下丘脑-垂体-上腺 (HPA) 和交感-上腺 (SAM) 轴,调节这种损伤.
- 减少认知灵活性显著介导压力对创造性原创性的负面影响.
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