在慢性间歇性缺氧引起的血管内皮损伤中,NAD+生物合成减少介导的线粒体功能障碍的特定作用

Z-T Fan1, L-P Dong, Y-H Niu

  • 1Clinical Biobank, The First Hospital of Hebei Medical University, Shijiazhuang, Hebei, China. songdongmei@hebmu.edu.cn.

概括

慢性间歇性缺氧 (CIH) 通过减少NAD+生物合成和引起线粒体功能障碍,损害内皮功能. 补充NAD+前体可能有助于治疗CIH诱导的血管损伤.

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