双甲诱导神经元亡并通过Nrf2/HO-1和Akt/mTOR通路在体外增强自

Yue Shen1, Xinying Li1, Hongyan Wang1

  • 1Neurodegeneration and Neuroregeneration Laboratory, Department of Basic Medicine, School of Medicine, Shaoxing University, Shaoxing, Zhejiang, China.

Toxicology
|November 25, 2023
PubMed
概括

双A (BPA) 通过增加氧化应激和亡,导致神经细胞死亡,同时破坏自和关键信号通路. 需要进一步的体内研究来证实这些神经毒性机制.

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