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Updated: Jul 9, 2025

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T-2毒素通过p53-cyclophilin D路径诱导 mitochondrial 功能障碍在红细胞中

Fang-Fang Yu1, Shui-Yuan Yu1, Lei Sun1

  • 1School of Public Health, Zhengzhou University, 100 Kexue Avenue, Zhengzhou, Henan 450001, China.

Journal of hazardous materials
|December 1, 2023
PubMed
概括

暴露于T-2毒素会通过损伤胆红细胞引起卡辛-贝克病. 这项研究揭示了T-2毒素激活了p53-cyclophilin D途径,导致线粒体功能障碍和细胞死亡.

关键词:
冠状细胞 (chondrocytes) 是一种细胞.线粒体功能障碍 线粒体功能障碍通过P53-CypD路径.这是一种T-2毒素.

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