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Updated: Jul 9, 2025

Gene-environment Interaction Models to Unmask Susceptibility Mechanisms in Parkinson's Disease
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Published on: January 7, 2014

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在Kynurenine 3-Monooxygenase (KMO) 基因和帕金森病患者之间的协会研究

Harysh Winster Suresh Babu1,2, Ajay Elangovan2, Mahalaxmi Iyer3,4

  • 1Human Molecular Cytogenetics and Stem Cell Laboratory, Department of Human Genetics and Molecular Biology, Bharathiar University, Coimbatore, 641 046, Tamil Nadu, India.

Molecular neurobiology
|December 1, 2023
PubMed
概括

帕金森病 (PD) 的风险受生活方式因素的影响. 对KMO基因的基因分析揭示了通过改变氨酸 (KYNA) 和3-氨 (3-HK) 水平,对晚期发病的PD有潜在保护性的突变.

关键词:
3 - 基氨 (3-HK) 是一种基氨.生物标志物 生物标志物京氨酸 (KYN) 是一种京氨酸3-单氧基酶 (KMO) 的使用生活方式因素 生活方式因素帕金森病 (PD) 是一种疾病.

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