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刺激性大脑状态的诱导控制了视觉皮层拓学的可塑性功能变化

Ulf T Eysel¹, Dirk Jancke²

¹Department of Neurophysiology, Ruhr University Bochum, 44780, Bochum, Germany. ulf.eysel@rub.de.

Brain structure & function

|December 2, 2023

概括

成年人视觉皮层的可塑性涉及细胞活动和兴奋能力的增加. 像跨磁刺激 (TMS) 这样的非侵入性刺激方法可以非侵入性地针对这种可塑性进行潜在的治疗应用.

科学领域:

神经科学是一个神经科学.
视觉系统可塑性视觉系统的可塑性
皮层重组 - 皮层重组

背景情况:

成年人视觉可塑性涉及到皮层电路的局部重塑.
这种重塑与自发和唤起的神经元活动的增加有关.
之前的研究表明,视网膜损伤会诱导视觉皮层的刺激性和功能重组.

研究的目的:

审查成人视觉皮层可塑性的原则.
讨论皮层刺激性增加和抑制性降低的作用.
为了证明视觉皮层重组的非侵入性向的证据.

主要方法:

电压敏感染料 (VSD) 成像用于跟踪皮质重组.
高频跨磁刺激 (TMS) 与VSD成像相结合.
在历史发展的背景下,对四项自己的研究进行了审查.

主要成果:

视网膜病变导致主视皮质的兴奋性增加和功能重组.
VSD成像可视化了皮质电路系统中病变诱导的重组.
通过TMS增强的兴奋性为非侵入性地重组定向图提供了基础.

结论:

在中视层面的LTP类皮质变化驱动成人视觉可塑性.
皮层刺激性增加是皮层模式的共同属性.
增加皮质刺激性的干预措施可能会推动感知和运动学习.

关键词:

皮层可塑性是指皮层的可塑性.功能性的重组组织.过度兴奋性过度兴奋性响应的可变性响应的可变性视网膜损伤视网膜损伤通过的磁性刺激 (TMS)视觉皮层的视觉皮层.

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