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Updated: Jul 9, 2025

Establishing a Mouse Model of a Pure Small Fiber Neuropathy with the Ultrapotent Agonist of Transient Receptor Potential Vanilloid Type 1
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Published on: February 13, 2018

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药物抑制的短暂的受体潜在离子通道安基林1对抗作用2-二马洛诺尼特利催剂诱导的皮肤损伤

Satyanarayana Achanta1, Narendranath Reddy Chintagari2, Shrilatha Balakrishna2

  • 1Center for Translational Pain Medicine, Department of Anesthesiology (S.A., B.L., S.-E.J.) and Department of Pharmacology and Cancer Biology (S.-E.J.), Duke University School of Medicine, Durham, North Carolina; Department of Pharmacology, Yale University School of Medicine, New Haven, Connecticut (N.R.C., S.B.); and Integrated Toxicology and Environmental Health Program (ITEHP), Nicholas School of the Environment, Duke University, Durham, North Carolina (S.-E.J.) satya.achanta@duke.edu sven.jordt@duke.edu.

The Journal of pharmacology and experimental therapeutics
|December 5, 2023
PubMed
概括

与普遍认为的相反,二二 (Chlorobenzalmalononitrile (CS)) 催气会导致持续的皮肤损伤. 针对TRPA1离子通道的治疗有效地减少了CS引起的皮肤损伤和疼痛.

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