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Updated: Jul 9, 2025

Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway
07:15

Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway

Published on: August 23, 2024

442

酸衍生物YPX-C-05通过PI3K/Akt/eNOS通路缓解高血压和血管功能障碍

Pan-Pan Pang1, Hao Sun2, Pei-Xia Yu2

  • 1School of Pharmaceutical Science and Yunnan Key Laboratory of Pharmacology for Natural Products, Kunming Medical University, Kunming 650500, China.

Vascular pharmacology
|December 5, 2023
PubMed
概括

新型HDAC抑制剂YPX-C-05通过激活PI3K/Akt/eNOS通路,显示出显著的血管扩张和抗高血压作用. 这项研究突出了其作为高血压新疗法药物的潜力.

关键词:
在 Akt/eNOS/NO 路径上.胺酸衍生物YPX-C-05 是一种胺酸衍生物.在高血压的高血压.血管功能 血管功能血管扩张是一种血管扩张.

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