α-2a:?

Melis Yavuz1, Serdar Akkol2, Filiz Onat3,4

  • 1Department of Pharmacology, Faculty of Pharmacy, Acibadem Mehmet Ali Aydinlar University University, Istanbul, Turkey.

Epilepsia open
|December 10, 2023
PubMed
概括

这项研究引入了使用α-2a上腺素受体 (α2AR) 激活的缺席状态的新老鼠模型. 阿提巴梅有效地减少了没有镇静作用的发作,这表明有针对性治疗的潜力.

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