[脱脂症和风湿性关节炎相关的间歇性肺病之间的相关性]
Qi Wu1,2, Yue Ming Cai1, Juan He1
1Department of Rheumatism and Immunology, Peking University Shenzhen Hospital; Shenzhen Key Laboratory of Immunity and Inflammatory Diseases, Shenzhen 518000, Guangdong, China.
概括
低密度脂蛋白胆固醇 (LDL-C) 是类风湿性关节炎相关间歇性肺病 (RA-ILD) 的风险因素,而高密度脂蛋白胆固醇 (HDL-C) 是保护性的. 较低的HDL-C水平与较早的ILD发病和RA患者的疾病活性增加相关.
科学领域:
- 类风湿病学 类风湿病学
- 肺部病理学 肺部病理学
- 心血管科学 心血管科学
更多相关视频
07:29Cell-free Biochemical Fluorometric Enzymatic Assay for High-throughput Measurement of Lipid Peroxidation in High Density Lipoprotein
Published on: October 12, 2017
9.3K
10:21Author Spotlight: Exploring the Role of Inflammation in the Co-occurrence of Primary Sjogren's Syndrome and Lung Adenocarcinoma
Published on: September 20, 2024
460
相关概念视频
01:20Chronic Obstructive Pulmonary Disease-II: Pathophysiology
2.8K
Chronic Obstructive Pulmonary Disease (COPD) pathophysiology is intricate and multifaceted, involving a complex interplay of physiological processes. Understanding these mechanisms is crucial for effectively managing and treating COPD. Here is an in-depth look at the critical elements in the pathophysiology of COPD:
Chronic Inflammation
Chronic Inflammation
2.8K
01:20The JAK-STAT Signaling Pathway
8.9K
Several cytokine receptors have tightly bound Janus kinase or JAK proteins attached at their cytosolic tail. Small signaling molecules such as cytokines, growth hormones, or prolactins bind to the cytokine receptors and initiate their dimerization. The dimerization brings the cytosolic JAKs together that trans-phosphorylate and activates each other. The activated JAKs now phosphorylate cytosolic tails of the cytokine receptors, which serve as binding sites for adaptor proteins such as SH2...
8.9K