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通过PI3K/AKT/mTOR路径调节来调节乳腺癌的进展

Mariam Naveed1, Ayesha Malik1, Hamza Anjum1

  • 1Centre of Excellence in Molecular Biology, University of the Punjab, 87-West Canal Road Thokar Niaz Baig , Lahore, 53700, Pakistan.

Biochemical genetics
|December 19, 2023
PubMed
概括

长非编码RNAMALAT1在巴基斯坦乳腺癌患者中被上调,并驱动瘤生长. 沉默MALAT1通过降低PI3K/AKT/mTOR通路的调节来抑制乳腺癌细胞的增殖和转移,这表明MALAT1是治疗点.

关键词:
乳腺癌 乳腺癌 乳腺癌长的非编码RNA可以编码.马拉特语 马拉特语在PI3K/AKT/mTOR路径中.

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