引起的脑炎作为抗N-甲基-d-阿斯巴酸受体脑炎的触发因素
Thomas Agerbo Gaist1, Anna Christine Nilsson2, Mette Scheller Nissen1
1Department of Neurology, Odense University Hospital, Odense, Denmark; Department of Clinical Research, University of Southern Denmark, Odense, Denmark.
Ticks and tick-borne diseases
|December 22, 2023
概括
感染性脑炎 (TBE) 可以引发N-甲基-d-酸盐受体 (NMDAR) 脑炎. 这一案例凸显了需要考虑TBE症状患者自身免疫性脑炎的必要性.
科学领域:
- 神经学 神经学
- 传染性疾病 传染性疾病
- 免疫学 免疫学 免疫学
背景情况:
- 传染性脑炎 (TBE) 是欧洲和亚洲日益严重的公共卫生问题.
- TBE通常会导致脑膜炎,因此需要意识到其潜在的并发症.
- 自身免疫性脑炎,特别是N-甲基-d-酸盐受体 (NMDAR) 脑炎,是一个诊断挑战.
更多相关视频
10:19High-throughput Flow Cytometry Cell-based Assay to Detect Antibodies to N-Methyl-D-aspartate Receptor or Dopamine-2 Receptor in Human Serum
Published on: November 23, 2013
16.3K
08:03Myelin Oligodendrocyte Glycoprotein MOG35-55 Induced Experimental Autoimmune Encephalomyelitis EAE in C57BL/6 Mice
Published on: April 15, 2014
82.1K
