系统性审查:在插入腹腔关节 (VPS) 后的神经缺陷
Rana Moshref1, Rafaa Ahmed Algethmi1
1Department of Neuroscience, King Faisal Specialist Hospital and Research Centre, Jeddah, Saudi Arabia.
Asian journal of neurosurgery
|December 28, 2023
概括
腹腔皮质突变 (VPS) 可以导致罕见的神经障碍,影响神经和大脑结构. 尽管VPS是常见的水头治疗方法,但需要仔细考虑.
科学领域:
- 神经外科 神经外科
- 神经学 神经学
- 医疗器械 医疗器械
背景情况:
- 脑水症通常需要腹腔关节 (VPS) 来控制内压力.
- 在VPS安置后的神经损伤是罕见的,但有记录的并发症.
研究的目的:
- 系统地审查和记录由心室关节 (VPS) 程序引起的神经缺陷.
- 为了确定与VPS相关的神经损伤的发生率和类型,VPS在脑水患者.
主要方法:
- 从1975年到2021年12月的PubMed和Cochrane数据库的系统审查.
- 包括标准:VPS,神经缺陷,人类参与者. 排除标准:其他造型,非人类受试者,感染.
主要成果:
- 在785名患者中,35名 (4.46%) 在VPS后经历了神经障碍.
- 神经 (VI,VII) 是最常见的 (44%),其次是脑干 (60%) 和深层脑结构损伤 (20%).
- 在36%的病例中,像被困的第四心室这样的潜在条件导致了赤字.
结论:
- 虽然VPS是一种标准的水头治疗方法,但必须承认潜在的神经并发症,尽管很少见.
- 意识到这些风险,包括神经和脑干损伤,对于患者的护理和管理至关重要.
关键词:
我们的VPS是VPS.神经学的缺陷 神经学的缺陷帕西斯·帕尔西斯 (palsies) 是一个残疾人.拖拉机拖拉机是什么意思腹腔上皮管外围突变 (ventriculoperitoneal shunt) 已经开始使用.更多相关视频
14:59Neuronavigation and Laparoscopy Guided Ventriculoperitoneal Shunt Insertion for the Treatment of Hydrocephalus
Published on: October 14, 2022
7.3K
04:33Spinal Hernia Repair and Cauda Equina Repositioning After Lumbar Decompression under Three-Dimensional Microscopy: A Case Report and Literature Review
Published on: November 8, 2024
391
相关概念视频
Aneurysm IV: Nursing Management
Vigilant monitoring for aneurysm rupture is essential for patients undergoing aortic surgery.Preoperative Nursing ManagementContinuously monitor the patient for manifestations of aneurysm rupture, such as pallor, weakness, tachycardia, hypotension, abdominal, back, groin, or periumbilical pain, changes in consciousness, and a pulsating abdominal mass. Regularly assess the patient's peripheral pulses.Instruct the patient to consume a clear liquid diet the day before surgery and administer...
Brain Abscess l: Introduction
A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...
Increased Intracranial Pressure ll: Pathophysiology
Increased intracranial pressure (ICP) refers to a potentially life-threatening rise in pressure inside the skull. This usually happens when there is a major change in the volume of brain tissue, blood, or cerebrospinal fluid (CSF) — the three components inside the skull. According to the Monro-Kellie doctrine, if the volume of one component increases, the volumes of the other components must decrease to maintain normal pressure. If this does not happen, ICP rises.The process often begins with...
Cerebral Edema ll: Pathophysiology
Vasogenic edema is a major form of cerebral edema characterized by abnormal accumulation of fluid in the brain’s extracellular space due to disruption of the blood–brain barrier (BBB). The BBB is a specialized structure composed of endothelial cells connected by tight junctions, supported by astrocytic endfeet and a basement membrane. Under normal conditions, it tightly regulates the movement of ions, proteins, and solutes between the bloodstream and brain parenchyma. When this barrier loses...
Hepatic Encephalopathy
DefinitionHepatic encephalopathy is a reversible neurologic syndrome that results from advanced liver dysfunction or portosystemic shunting. It leads to disturbances in cognition, behavior, and motor function due to the brain’s exposure to gut-derived toxins that the liver fails to detoxify.EtiologyThis condition develops either in the setting of acute fulminant hepatitis or progressively during chronic liver disease, such as cirrhosis and portal hypertension. Portosystemic shunting—including...
