在游泳运动员中,缺氧的外周化学反射刺激依赖于心肺呼吸系统的合性下降
David C Andrade1, Alexis Arce-Álvarez2, Camila Salazar-Ardiles1,3
1Exercise Applied Physiology Laboratory, Centro de Investigación en Fisiología y Medicina de Altura (FIMEDALT), Departamento Biomedico, Facultad de Ciencias de la Salud, Universidad de Antofagasta, Antofagasta, Chile.
Physiological reports
|January 9, 2024
概括
游泳者表现出心肺呼吸器合的减少和低氧呼吸器反应的减少. 这项研究研究了游泳者在低氧刺激期间使用时间变化的连贯性分析来研究心肺呼吸系统的合.
科学领域:
- 心血管呼吸系统生理学
- 运动科学运动科学
- 运动医学运动医学
背景情况:
- 游泳运动员表现出自主反应的变化,包括减少交感激活和呼吸系统对缺氧的反应.
- 游泳者的外周缺氧反应减弱可能导致心肺呼吸系统合功能受损.
研究的目的:
- 为了测试游泳者由于其拙的缺氧外周反应而减少了心肺呼吸合的假设.
- 在训练有素的游泳者中量化评估心肺呼吸系统合,与在正常和缺氧条件下的对照组相比较.
主要方法:
- 采用波束波段时间变化的光谱连贯性分析来检查呼吸频率 (Rf) 和心率 (HR) 之间的关系.
- 在诺莫克西亚和急性缺氧化学反射激活 (100% N2 吸入) 期间评估心肺呼吸系统合.
- 招募了12名训练有素的游泳者和12名对照对象.
主要成果:
- 与对照组相比,游泳者表现出明显减少的缺氧呼吸反应 (p < 0.001).
- 在缺氧期间 (p < 0.012) 观察到游泳者下部心肺呼吸系统合 (LF切断连贯性).
- 在N2吸入过程中,群体之间的 ΔVE/ΔVO2 和 ΔVE/ΔVCO2/ΔSpO2 的显著差异.
结论:
- 游泳者表现出减少的化学反射控制,导致低氧刺激期间心肺呼吸系统合减少.
- 这些发现支持了受过训练的游泳者心肺呼吸系统合障碍的假设.
- 表明对水上运动员呼吸和心血管调节的潜在影响.
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