在中部部份的阿基里斯肌病症中,不能假定足部曲器的软弱和疼痛敏感性
Lauren K Sara1,2, Savannah B Gutsch2, Marie Hoeger Bement2
1Harvard Medical School, Cambridge, MA, USA.
Exercise, sport & movement
|January 15, 2024
概括
中部部分阿基里斯肌病 (AT) 不会影响脚下曲的强度或功率. 然而,预测强度的机制不同,收缩功能是AT的关键,AT患者的全身疼痛敏感性降低.
科学领域:
- 生物力学 生物力学
- 神经肌肉功能 神经肌肉功能
- 疼痛科学 疼痛科学
背景情况:
- 中部部分阿基勒斯肌病 (AT) 是一种常见的肌肉骨疾病.
- 了解底层的神经肌肉和疼痛机制对于有效治疗至关重要.
研究的目的:
- 评估与对照组相比,AT患者的最大力量和功率.
- 在最大和引起的收缩期间调查神经驱动和收缩功能.
- 为了确定疼痛,神经驱动和收缩功能是否解释AT的强度差异.
主要方法:
- 28名参与者 (14名AT,14名对照人) 进行了足部 flexors的最大自愿收缩 (MVCs).
- 神经刺激量化了神经驱动和收缩性质.
- 压力疼痛值评估了阿基里斯肌,小腿和上的疼痛敏感性.
主要成果:
- 在AT和对照组之间,脚部屈曲强度或功率没有显著差异.
- 神经驱动和收缩功能在各组之间没有差异.
- 收缩功能预测了AT的最大强度 (P=0.001),而神经驱动器预测了对照的强度 (P=0.02).
- 患有AT的个体在上梯形上表现出更高的压力疼痛值 (P=0.02).
结论:
- 在中部部位阿基里斯肌病症的个体中,足部屈曲的强度和功率是保留的.
- 最大的强度背后的机制不同,在AT中,收缩功能更为关键.
- 在AT患者中,全身疼痛敏感性降低,这表明更广泛的疼痛调节变化.
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