Jang Eun Lee1, Karen Wilcox2, Kenneth A Jacobson3

  • 1Department of Medicine, Cardiovascular Division, Philadelphia, Pennsylvania.

Drug development research
|January 18, 2024
PubMed
概括

短暂缺血通过激活腺受体 (A1和A3) 和KATP通道来保护心脏细胞. 这种预先条件的现象减少了持续性缺血期间的损伤,保持ATP和细胞活力.

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