在登山者中缺氧和代谢性脑损伤后的康复
Henry Han Chen1, Charnette Lercara2, Vincent Lee3
1Department of Physical Medicine and Rehabilitation, Burke Rehabilitation Hospital, White Plains, New York, USA henchen@burke.org.
BMJ case reports
|January 18, 2024
概括
低水平 (低血症) 的快速纠正可能导致脑损伤. 抗氧化剂补充剂在改善患者的功能方面表现有前途,患者患有额外质髓溶解和延迟后缺氧性白细胞大脑病变.
科学领域:
- 神经学 神经学
- 内部医学 内部医学
- 关键护理医学 关键护理医学
背景情况:
- 严重的低血症的快速纠正可能会导致透性脱髓化综合征,包括额外质髓溶解 (EPM).
- 延迟后缺氧性白细胞大脑病变 (DPHL) 是缺氧性脑损伤后的一种罕见的神经并发症.
- 无论是EPM还是DPHL,都存在诊断和治疗方面的挑战,往往治疗选择有限.
更多相关视频
04:43Author Spotlight: Advancing Upper Limb Rehabilitation in Patients with Right Hemisphere Damage Using Assisted Active Exercise
Published on: February 9, 2024
1.1K
10:13Experimental Models to Study the Neuroprotection of Acidic Postconditioning Against Cerebral Ischemia
Published on: July 31, 2017
7.5K
相关概念视频
Acute Respiratory Failure-II
238
Type I Respiratory Failure, or hypoxemic respiratory failure, occurs when the partial pressure of oxygen (PaO2) in arterial blood falls below 60 mmHg while breathing room air without a corresponding increase in arterial carbon dioxide levels (PaCO2). This condition highlights a significant impairment in the lungs' capacity to oxygenate the blood.
The underlying physiological abnormalities that contribute to hypoxemic respiratory failure include:
The underlying physiological abnormalities that contribute to hypoxemic respiratory failure include:
238
Acute Respiratory Failure-III
187
Hypercapnic respiratory failure, also known as Type 2 or ventilatory respiratory failure, is a severe condition characterized by the body's inability to effectively remove carbon dioxide (CO2) from the bloodstream. It leads to an arterial CO2 pressure (PaCO2) exceeding 45 mmHg and a blood pH above 7.35. This situation indicates that the body's ventilatory demand, or the ventilation needed to maintain normal PaCO2 levels, surpasses its supply or the maximum gas flow achievable without...
187
