用ADA生物相似药物治疗后的Morphea的发展:一个案例报告
Aliki I Venetsanopoulou1, Konstantina Mavridou2, Eleftherios Pelechas1
1Department of Rheumatology, Faculty of Medicine, School of Health Sciences, University of Ioannina, Ioannina, Greece.
Current rheumatology reviews
|January 20, 2024
概括
瘤亡因子α抑制剂 (TNFi) 具有悖论意义,可以导致皮肤疾病 - - 形态. 一名接受阿达利穆马布生物类似药的患者出现了形,在停止服用药物后得到缓解.
科学领域:
- 类风湿病学 类风湿病学
- 皮肤病学 皮肤病学
- 免疫学 免疫学 免疫学
背景情况:
- 瘤缩因子α (TNFα) 是类风湿性关节炎 (RA) 等炎症性疾病的关键.
- TNFα 抑制剂 (TNFi) 已经改变了 RA 治疗,但可以引起自身免疫性副作用.
- 矛盾的皮肤反应,包括形态变异 (局部性硬皮),是已知的TNFi并发症.
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