奥克拉托辛A刺激了从培养的人类微质中释放IL-1β,IL-18和CXCL8

Irene Tsilioni1, Theoharis C Theoharides2

  • 1Laboratory of Molecular Immunopharmacology and Drug Discovery, Department of Immunology, Tufts University School of Medicine, Boston, MA 02111, USA.

Toxicology
|February 1, 2024
PubMed
概括

奥克拉托克素A (OTA) 通过激活微质,释放炎症性细胞因子,触发神经炎症. 丁醇可能会抑制这种激活,可能会减轻相关的神经精神疾病.