在有细分睡眠模式的成年人中增加了氧化应激生物标志物
Ibtisam Al Lawati1, Fahad Zadjali2, Mohammed A Al-Abri3
1Department of Support Sciences, Oman College of Health Sciences, Muscat, Bousher, Sultanate of Oman.
概括
分段睡眠模式,如双相和多相睡眠,与增加的氧化应激生物标志物有关,包括甲和基底氧化能力. 单相睡眠与较高的总谷氨水平有关.
科学领域:
- 睡眠医学 睡眠医学
- 生物标志物 生物标志物
- 氧化压力是一种氧化压力.
背景情况:
- 成人的睡眠模式在很大程度上不同.
- 炎症和氧化应激生物标志物是健康的关键指标.
- 了解睡眠与这些生物标志物之间的联系对于公共健康至关重要.
研究的目的:
- 研究不同睡眠模式 (单相,双相,多相) 与成年人关键炎症和氧化应激生物标志物之间的关联.
- 为了确定睡眠碎片化是否会影响特定的生物标志物,如高灵敏度C反应蛋白,甲,总谷和基底氧化能力.
主要方法:
- 这是一项涉及321名年龄在18-59岁之间的成年人的横截面研究.
- 参与者在一周内佩戴了动图仪,以记录睡眠模式 (单相,双相,多相).
- 禁食血液样本被分析为炎症和氧化应激生物标志物.
主要成果:
- 没有发现睡眠时间/午睡时间与高灵敏度C反应蛋白,甲,全谷氨或基底氧化能力之间的相关性.
- 分段睡眠模式 (双相和多相) 与氧化应激生物标志物增加有显著关联.
- 多相睡眠与较高的基底氧化能力相关 (P=.023),双相睡眠与较高的甲基 (P=.036),单相睡眠与较高的总谷氨 (P=.046).
结论:
- 细分睡眠,以双相和多相模式为特征,与血清甲酸和基本氧化能力的升高有关.
- 建议进行进一步的研究,以探索这些氧化应激生物标志物的心脏代谢影响,在睡眠模式细分的个体中.
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