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CD36抑制通过AKT-mTOR通路减少非小细胞肺癌的发展

Hui Liu1, Wentong Guo2, Tianxiang Wang2

  • 1Department of Respiratory and Critical Care Medicine, The Second Affiliated Hospital of Anhui Medical University, Hefei, China.

Cell biology and toxicology
|February 6, 2024
PubMed
概括

高脂肪饮食通过增加CD36受体活性来加速非小细胞肺癌 (NSCLC) 的生长. 用皮塔瓦斯塔丁抑制CD36显示出治疗NSCLC的前景,特别是在饮食影响的情况下.

关键词:
在 AKT/mTOR 路径上.CD36 CD36 CD36 CD36 CD36 CD36 CD36 CD36 CD36 CD36 CD36 CD36 CD36 CD36在FFA和FFA之间.肺癌是一种肺癌.皮塔瓦斯塔丁是一种药物.

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