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Updated: Jul 3, 2025

Activation and Measurement of NLRP3 Inflammasome Activity Using IL-1β in Human Monocyte-derived Dendritic Cells
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SERTAD1通过限制NLRP3的多基化,启动NLRP3介导的炎症酶激活

Jihoon Ha1, Minbeom Kim1, Jin Seok Park1

  • 1Department of Biological Sciences, Sungkyunkwan University, Suwon 16419, Republic of Korea.

Cell reports
|February 11, 2024
PubMed
概括

通过控制多比基因化,SERTAD1调节NLRP3炎症酶激活. 这种适应蛋白减少了自身免疫性疾病的炎症,提供了潜在的治疗点.

关键词:
CP: 免疫学 免疫学在CUL1中,CUL1是CUL1的第一个类型.与K63结合的多联化.在NLRP3炎症酶体中,NLRP3炎症这就是SERTAD1的原因.自身免疫性疾病 自身免疫性疾病

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