睡眠症:一个概念的过去,现在和未来的演变
Journal of the American Dental Association (1939)
|February 16, 2024
概括
越来越多的人认为睡眠 Bruxism (SB) 源于中枢神经系统的因素,而不是牙问题. 研究表明,自主神经系统,遗传学和并发症在动运动中起着关键作用.
科学领域:
- 神经科学是一个神经科学.
- 睡眠医学 睡眠医学
- 牙科研究 牙科研究
背景情况:
- 自20世纪60年代以来,对睡眠性 Bruxism (SB) 的理解有了显著的进化.
- 早期的假设集中在外围因素上,如牙堵塞,但这些基本上被抛弃了.
- 随着时间的推移,关于SB的定义,病理生理学和管理的许多理论被提出.
研究的目的:
- 审查关于睡眠布鲁克斯症的科学文献.
- 确定对SB的潜在原因和促成因素的不断变化的理解.
- 突出SB研究中的范式转变,从外围机制到中心机制.
主要方法:
- 在包括PubMed,Google Scholar,Web of Science,Embase和Ovid MEDLINE在内的主要电子数据库中进行了全面的文献搜索.
- 搜索涵盖了1961年1月至2023年5月发表的文章.
- 总共有4,612篇文章被确定,其中312篇在删除重复和无关研究后被选择进行详细审查.
主要成果:
- 显而易见的是,在理解SB病原体的过程中,从外围因素 (例如,牙封闭) 转向中心因素的理解发生了明显的转变.
- 自主神经系统和中央模式生成器越来越多地涉及到布鲁克斯运动的起源.
- 牙科界对了解导致SB的因素保持着强烈的兴趣.
结论:
- SB的神经生理学主要与中心而不是外围因素有关.
- 越来越多的证据支持自主神经系统,遗传学和并发症在SB发育中的作用.
- 当前的科学文献驳斥了牙堵塞在引起牙病的作用,表明其定义和病因学的范式转变.
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