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Updated: Jul 3, 2025

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在患有遗传IFN-γ缺乏症的患者中进行复合IFN-γ1b治疗

Jérémie Rosain1,2,3, Ayca Kiykim4, Alexandre Michev5,6

  • 1Laboratory of Human Genetics of Infectious Diseases, Necker Branch, Necker Hospital for Sick Children, INSERM U1163, Paris, France. jeremie.rosain@institutimagine.org.

Journal of clinical immunology
|February 16, 2024
PubMed
概括

一名患有孟德尔易感菌菌病 (MSMD) 的新患者因一种新型IFNG变异而出现了细胞内但未分泌的干扰素- (IFN-γ). 重组IFN-γ1b治疗成功控制了真菌菌感染,为干细胞移植提供了替代方案.

关键词:
在BCG中,BCG是BCG.免疫的天生的错误.干扰素 - 玛的干扰素.这是一种Mycobacterium细菌.

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Tumor Necrosis Factor (TNF), a proinflammatory cytokine, contributes significantly to the inflammation seen in Crohn's disease. It exists as soluble TNF and membrane-bound TNF, with actions mediated through TNF receptors (TNFR). TNFR activation leads to the release of proinflammatory cytokines, T-cell activation, collagen production, and leukocyte migration, all contributing to inflammation in Crohn's disease. Anti-TNF monoclonal antibodies, namely infliximab (Remicade), adalimumab...
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