[非伤寒型沙门氏菌中诺隆耐药性的分子机制]

J J Luo1, S H Zeng1, Y L Huang1

  • 1Department of Laboratory Medicine, the Fifth Affiliated Hospital of Southern Medical University, Guangzhou 510900, China.

概括

非伤寒型沙门氏菌 (NTS) 呈现出显著的醇耐药性,主要是由qnrS1基因和gyrA突变驱动的. 这种耐药性会影响常见的抗生素,如西普罗夫洛克萨和纳利迪克酸,需要进一步监测.