脊髓缩症第一部分:文献综述
Saverio Colonna1, Corrado Borghi1
1Osteopathic Spine Center Education (OSCE), Spine Center, Bologna, ITA.
Cureus
|February 26, 2024
概括
关节炎 (RA),或指关节骨关节炎,是一种导致疼痛和限制功能的退行性疾病. 多种模式的保守治疗,包括运动和手动治疗,有效地减少疼痛和改善RA患者的机械.
科学领域:
- 整形外科 整形外科 整形外科
- 类风湿病学 类风湿病学
- 物理疗法物理治疗
背景情况:
- 脊髓缩症 (RA) 或梯形甲手腕骨关节炎是一种退行性关节疾病,影响指的第一个甲手腕关节.
- 关节炎在绝经后的女性中很普遍,与衰老有关,涉及由荷尔蒙和机械因素影响的带和肌肉结构.
- 了解指关节的生物力学,稳定性和促成因素对于有效的治疗管理至关重要.
研究的目的:
- 为治疗师提供管理形甲手关节不稳定的基本问题的概述.
- 探索带,肌肉和解剖学变体在指关节退化中的作用.
- 为了强调梯形甲手关节稳定性和一致性的重要性.
主要方法:
- 临床评估包括疼痛评估,关节运动检查和触摸.
- 诊断测试,如磨砂测试和杆测试,以及放射检查 (伊顿-利特勒分类).
- 对保守治疗方式的审查:骨架,治疗炼和手动疗法 (神经动力学,卡尔博恩,穆利根,梅特兰技术).
主要成果:
- 关节炎表现为基本指疼痛,握力降低和功能限制,可能会发展为持续的不适和睡眠障碍.
- 慢性RA可以导致关节硬,形 (例如"Z指"),肌肉缩,显著影响日常活动.
- 结合关节调动,神经调动和运动的多式保守干预措施在RA患者的疼痛减轻方面表现出有效性.
结论:
- 保守的管理是根茎的首要方法,旨在减轻疼痛,减少关节压力和增强功能.
- 综合各种治疗技术的多式保守方法在治疗疼痛和改善关节机制方面是有效的.
- 当保守疗法无法提供足够的缓解时,就会考虑进行手术干预.
相关概念视频
The Spinal Cord
27.5K
The spinal cord is the body’s major nerve tract of the central nervous system, communicating afferent sensory information from the periphery to the brain and efferent motor information from the brain to the body. The human spinal cord extends from the hole at the base of the skull, or foramen magnum, to the level of the first or second lumbar vertebra.
27.5K
Spinal Cord: Cross-sectional Anatomy
5.7K
The cross-sectional anatomy of the spinal cord offers a detailed view of its complex structure and function within the central nervous system. At the core of the spinal cord lies the gray matter, characterized by its butterfly or "H"-shaped appearance in cross-section. This central region is enveloped by white matter, with the overall structure divided into symmetrical halves by the dorsal median sulcus and the ventral median fissure.
Gray Matter and its Components
Central to the gray matter is...
Gray Matter and its Components
Central to the gray matter is...
5.7K
Multiple Sclerosis l: Introduction
20
Multiple sclerosis is a chronic autoimmune disease of the central nervous system (CNS) that affects the brain, spinal cord, and optic nerves. It is an inflammatory demyelinating disorder and a leading cause of neurological disability in young adults.EpidemiologyMS commonly begins between 20 and 40 years of age and is twice as common in women. Its exact cause remains unclear, but genetic susceptibility contributes, with higher risk in first-degree relatives and identical twins. A greater...
20
Huntington Disease l: Introduction
110
Huntington disease or HD is a progressive, fatal neurodegenerative disorder inherited in an autosomal dominant pattern.PathophysiologyIt is caused by expansion of the CAG trinucleotide repeat in the HTT gene on chromosome 4 (4p16.3), producing an abnormal huntingtin protein with an expanded polyglutamine tract. This misfolded protein disrupts cellular function, leading to neuronal death. Normal alleles have ≤26 repeats, 27–35 are intermediate (risk of expansion), 36–39 show...
110
Dementia l: Introduction
35
Dementia is an acquired, progressive syndrome characterized by a decline in multiple cognitive domains severe enough to impair daily functioning and reduce independence. Although memory loss is a central feature, the diagnosis requires additional deficits involving language, executive function, visuospatial skills, judgment, calculation, or abstract reasoning. These cognitive impairments reflect underlying neurodegenerative or vascular processes that gradually disrupt neuronal networks...
35
Secondary Spinal Cord Injury llI: Pathophysiology
52
Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...
52


