血流限制增加了运动引起的压力疼痛值,超过了重复和努力匹配的条件
William B Hammert1, Jun Seob Song1, Yujiro Yamada1
1Department of Health, Exercise Science and Recreation Management. Kevser Ermin Applied Physiology Laboratory, The University of Mississippi, University, MS, USA.
Journal of sports sciences
|February 27, 2024
概括
低负荷运动的血液流量限制 (BFR) 显著增强了运动诱导的缺血症,系统地增加了压力疼痛值. 这种影响是BFR特有的,并没有改变缺血性疼痛敏感性.
科学领域:
- 运动生理学 运动生理学
- 疼痛管理 疼痛管理
- 康复科学 康复科学 康复科学
背景情况:
- 运动诱导的低痛感是一种记录良好的现象.
- 由于其潜在的益处,血液流量限制 (BFR) 训练越来越多地被利用.
- 对于BFR对低负荷运动诱导的低质症的具体影响需要进一步阐明.
研究的目的:
- 研究低负荷 (LL) 阻力运动与血液流量限制 (BFR) 结合对运动诱导的低痛症的影响.
- 为了比较LL+BFR运动与LL运动,LL运动与努力匹配的对照,以及非运动对照的减痛效应.
主要方法:
- 44名参与者接受了四个实验条件:LL+BFR练习,LL练习,LL+EFFORT练习和时间匹配的控制.
- 低负荷阻力运动是在一次重复最大 (1RM) 的30%进行的,重复基于个体肌肉耐力.
- 干预前和后测量了压力疼痛值 (PPT);干预后评估了缺血性疼痛值和耐受性.
主要成果:
- 与LL练习,LL+EFFORT练习和对照条件相比,LL+BFR练习导致上半身和下半身PPT显著增加.
- 在任何疾病中都没有观察到缺血性疼痛值或容忍度的显著变化.
- 减痛效应似乎是BFR干预的特征,因为与努力匹配的自由流条件没有产生类似的结果.
结论:
- 亚最大阻力运动与血液流量限制相结合,有效地产生系统性缺血症.
- 观察到的减痛效应很可能归因于BFR成分,而不是单独的运动强度或努力.
- 在运动干预后,BFR可能是增强疼痛调节的有价值补充.
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