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线粒体KATP通道介导的自促使小鼠的血管功能障碍产生血管酶II诱导的血管功能障碍

Xue-Min Yin1, Yi-Yi Song1, Wen-Yi Jiang1

  • 1Department of Physiology, Medical College of Soochow University, 199 Ren-Ai Road, Dushu Lake Campus, Suzhou Industrial Park, Suzhou 215123, PR China.

Nutrition, metabolism, and cardiovascular diseases : NMCD
|February 28, 2024
PubMed
概括

线粒体KATP通道和自细胞在血管激素II (Ang II) 诱导的高血压和动脉样硬化中起着关键作用. 抑制这些通路可以预防血管功能障碍和相关疾病.

关键词:
ангиотензин II 的使用.动脉样硬化是一种动脉样硬化.自自是一种自的过程.在高血压的高血压.线粒体K ((ATP) 通道

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