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Updated: Jul 21, 2026

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M2 微质衍生外体通过外体 miR-124-3p 保护免受谷氨酸诱导的HT22细胞损伤

Lan Zhu1, Limei Ma1, Xin Du1

  • 1Department of Emergency and Critical Care Medicine, The Second Affiliated Hospital of Soochow University, 1055 Sanxiang Road, Suzhou, Jiangsu, 215004, People's Republic of China.

Molecular neurobiology
|March 3, 2024
PubMed
概括

来自M2微质细胞的外生体通过输送miR-124-3p来保护神经元免受谷氨酸损伤,减少细胞亡和氧化应激. 这为败血症相关脑病变提供了一种新的治疗策略.

关键词:
细胞灭亡 (apoptosis) 是一种死亡的过程.外基因组是外基因组的组成部分.巨细胞是一个巨细胞.微质细胞中的微质细胞神经保护是一种神经保护.氧化应激是一种氧化应激.在Rock1上,你会看到Rock1.在Rock2上,我一直在努力.败血症相关的脑病变 - - 败血症相关的脑病变在 miR-124-3p 中使用.

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