在败血症中,BAP31通过NF-κB信号通路促进内皮细胞和巨细胞之间的粘附

Jiawei He1, Danyang Jing1, Shen Zhao1

  • 1Department of Critical Care Medicine, Beijing Friendship Hospital, Capital Medical University, Beijing, People's Republic of China.

概括

B细胞受体关联蛋白31 (BAP31) 通过增加内皮细胞粘附分子来促进败血症. 在败血症模型中减少BAP31通过抑制炎症信号通路来降低死亡率和器官损伤.

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