阻断阿里尔碳化合物受体限制了欧梅普拉诱导的慢性病

Nan Sun1,2, Yimeng Zhang2, Lin Ding2

  • 1Laboratory of Basic Medical Sciences, Qilu Hospital of Shandong University, Jinan, Shandong, China.

Journal of molecular medicine (Berlin, Germany)
|March 7, 2024
PubMed
概括

长期使用奥梅普拉可以通过激活细胞中的酸受体 (AHR) 途径引起慢性病 (CKD). 阻止这种AHR-CYP1A1通路为梅诱导的损伤提供了潜在的治疗策略.

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