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Updated: May 12, 2026

Stimulation of Notch Signaling in Mouse Osteoclast Precursors
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通过GSK3β/β-catenin信号通路,KDELR2促进骨髓中酶体干细胞的骨质分化

Xiaoyong Wu1,2,3,4, Weijun Zhang1,2,3,4, Long Long1,2,3,4,5

  • 1Department of Orthopedic Surgery of the Second Affiliated Hospital, School of Medicine, Zhejiang University, No. 88, Jiefang Road, Hangzhou 310009, China.

Cell and tissue research
|March 12, 2024
PubMed
概括

Lys-Asp-Glu-Leu (KDEL) 细胞内网膜蛋白质保留受体2 (KDELR2) 促进骨愈合. 通过调节GSK3β/β-catenin通路,KDELR2可以增强骨质分化和骨折修复.

关键词:
在GSK3β/β-catenin中.在KDELR2R2中使用.骨质发生过程 (osteogenesis)在mBMSCs.

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相关概念视频

TGF - β Signaling Pathway01:16

TGF - β Signaling Pathway

The TGF-β signaling pathway regulates cell growth, differentiation, adhesion, motility, and development. TGF-β ligands that induce TGF-β signaling are synthesized in their latent form. Several proteases or cell surface receptors such as integrins act upon the latent form, releasing the active ligand. There are three types of mammalian TGF-βs: (TGF-β1, TGF-β2, and TGF-β3) that bind as homodimers or heterodimers to TGF-β receptors. The TGF-β receptors are of three kinds RI, RII, and RIII. The RI...