IL-6信号驱动脂肪组织巨细胞的自我更新和替代激活

Jan Ackermann1,2, Lilli Arndt1,2, Janine Fröba1

  • 1Institute of Anatomy, Leipzig University, Leipzig, Germany.

PubMed
概括

干白素-6 (IL-6) 减少炎症并维持脂肪组织中的巨细胞增殖,独立于IL-4Rα信号传递. 这表明IL-6在控制与肥胖相关的炎症和巨细胞动态方面发挥着关键作用.

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