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Updated: Jun 30, 2025

Induction of Murine Intestinal Inflammation by Adoptive Transfer of Effector CD4+CD45RBhigh T Cells into Immunodeficient Mice
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NOD2和TLR2之间的交叉声抑制了TLR2介导的实验性大肠炎的发展

Natsuki Okai1, Yasuhiro Masuta1, Yasuo Otsuka1

  • 1Department of Gastroenterology and Hepatology, Kindai University Faculty of Medicine, 377-2 Ohno-Higashi, Osaka-Sayama, Osaka 589-8511, Japan.

Journal of clinical biochemistry and nutrition
|March 21, 2024
PubMed
概括

核酸结合寡合化域2 (NOD2) 和托尔类受体2 (TLR2) 的交叉影响大肠炎. NOD2 缺乏会使结肠炎恶化,而TLR2 缺乏会保护,揭示了它们在肠道炎症中的对立作用.

关键词:
在ATG16L1中使用.在NOD2中,NOD2是NOD2.这就是TLR2的特点.大肠炎是一种大肠炎.

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Development of an Antigen-driven Colitis Model to Study Presentation of Antigens by Antigen Presenting Cells to T Cells06:57

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T Cell Types and Functions01:24

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When T cells with CD4 markers are activated, they give rise to two types of effector cells: helper T cells and regulatory T cells. Meanwhile, T cells with CD8 markers differentiate into effector cytotoxic T cells. The differentiation of CD4 T cells into helper T cell subsets, such as Th1, Th2, and Th17 cells, is dependent on the antigen type, antigen-presenting cell, and regulatory cytokines.
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