脑卒中后失足症的神经康复:生理学和病理生理学
Ayodele Sasegbon1, Ivy Cheng1,2,3, Shaheen Hamdy1
1Division of Diabetes, Endocrinology and Gastroenterology, School of Medical Sciences, Centre for Gastrointestinal Sciences, Faculty of Biology, Medicine and Health, Salford Royal Foundation Trust, University of Manchester, Manchester, UK.
The Journal of physiology
|March 22, 2024
概括
吞涉及复杂的肌肉和神经控制. 本综述探讨了中风后的消化不良 (PSD),其原因,恢复机制和治疗方法.
科学领域:
- 神经科学是一个神经科学.
- 胃肠病学 胃肠病学
- 神经学 神经学
背景情况:
- 吞是一种复杂的神经肌肉过程,由大脑干,皮质和小脑控制.
- 消化不良或吞障碍可能是影响神经肌肉结构的各种疾病的结果.
- 脑卒中后消化障碍 (PSD) 是一种普遍且经过充分研究的形式,可以作为理解吞障碍的模型.
研究的目的:
- 探索正常吞和中风后吞障碍的神经解剖过程.
- 为了阐明神经可塑性恢复在中风后失足症的机制.
- 审查目前和新兴的治疗持续性消化障碍的方法.
主要方法:
- 关于吞神经解剖学和生理学的动物和人类研究的综述.
- 分析中风诱导的消化障碍机制.
- 检查神经可塑性和治疗性干预的治疗功能障碍症.
主要成果:
- 卒中会破坏神经控制吞的过程,导致吞障碍.
- 自然的神经可塑性促进了中风后吞功能的恢复.
- 对于持续性消化不良存在各种治疗方法,新兴的神经调节选择.
结论:
- 了解吞的神经解剖学对于解决吞障碍至关重要.
- 神经可塑性提供了恢复的潜力,但有针对性的治疗往往是必要的.
- 对神经调节治疗的进一步研究对治疗持续性消化不良有希望.
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