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Updated: Jun 30, 2025

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介乐金-37:在自体主导多囊性病中是一种新的治疗标

Bo Yang1, Steven D Crowley2

  • 1Division of Nephrology and Endocrinology, Naval Medical Center of PLA, Naval Medical University, Shanghai, People's Republic of China.

Kidney international
|March 22, 2024
PubMed
概括

介质素-37是一种新型细胞因子,通过激活中的免疫细胞来抑制自体主导多囊性病 (ADPKD) 的发病. 这一发现突出了ADPKD潜在的新疗法.

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Tumor Necrosis Factor (TNF), a proinflammatory cytokine, contributes significantly to the inflammation seen in Crohn's disease. It exists as soluble TNF and membrane-bound TNF, with actions mediated through TNF receptors (TNFR). TNFR activation leads to the release of proinflammatory cytokines, T-cell activation, collagen production, and leukocyte migration, all contributing to inflammation in Crohn's disease. Anti-TNF monoclonal antibodies, namely infliximab (Remicade), adalimumab...
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