运动后低血压的核心和外围机制:一个范围审查
Iedda A Brasil1, José Cristiano P L Silva1,2, Linda S Pescatello3
1Graduate Program in Exercise and Sport Sciences, University of Rio de Janeiro State, Rio de Janeiro, Brazil.
Journal of hypertension
|March 25, 2024
概括
运动后低血压 (PEH) 降低了运动后的血压. 虽然与自主控制有关,但局部血管扩张剂可能更为关键. 需要进一步的研究来澄清PEH机制.
科学领域:
- 运动生理学 运动生理学
- 心血管健康 心血管健康
- 高血压管理 高血压管理
背景情况:
- 运动后低血压 (PEH) 是运动后血压显著下降.
- PEH对慢性降血压和预测运动训练反应具有临床意义.
- 驱动PEH的精确生理机制仍然不完全理解.
研究的目的:
- 对现有的关于运动后低血压机制的文献评论进行范围审查.
- 综合当前对促进PEH的中心和外围生理系统的知识.
- 发现对PEH机制的理解差距,并指导PEH机制的未来研究方向.
主要方法:
- 在主要的科学数据库 (PubMed, Web of Science, Scopus, CINAHL, Cochrane, SportDiscus) 进行了全面的文献搜索,截至2023年1月.
- 该审查包括21篇文献评论:13篇叙述,4篇系统 (102个主要试验) 和4篇元分析 (75个主要试验,1566名参与者).
- 机制被分为中央 (自主神经系统,巴罗反射,心脏) 和外围 (血管,血液动力学,幽默,脏) 的类别.
主要成果:
- PEH通常与自主控制的改变有关,导致心脏输出减少或血管扩张.
- 有证据表明,在引起PEH时,重点可能从自主控制转向局部血管扩展因子.
- 各种生理因素对PEH的具体贡献尚不清楚,特别是在不同的运动类型和人群中.
结论:
- 虽然自主神经系统的变化有关,但局部血管扩展机制可能在PEH中发挥更重要的作用.
- 需要进一步的综合研究来阐明PEH的复杂机制.
- 了解PEH机制对于优化运动作为抗高血压疗法至关重要,特别是对于临床相关的血压降低.
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