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Updated: Jun 29, 2025

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m6A修改促进EMT和割抵抗性前列腺癌的转移通过UpregulatingNFIB

Feng Shu1, Hao Liu2, Xiaohui Chen1

  • 1Department of Clinical Laboratory, The Fifth Affiliated Hospital of Sun Yat-sen University, Zhuhai, Guangdong, China.

Cancer research
|March 27, 2024
PubMed
概括

核因子I/B (NFIB) 通过促进表皮细胞到介质细胞转变 (EMT) 来驱动雄激素受体阴性化耐性前列腺癌 (CRPC) 的转移. 准N6-甲基氨酸 (m6A) /NFIB通路为CRPC治疗提供了一个新的策略.

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