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Updated: Jun 29, 2025

Therapeutic Evaluation of Fecal Microbiota Transplantation in an Interleukin 10-Deficient Mouse Model
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缺乏Cmtm4会通过诱导肠道失调和S100a8/9表达来加剧结肠炎

Qiao Meng1, Jing Ning1, Jingjing Lu2

  • 1Department of Gastroenterology, Peking University Third Hospital, Beijing 100191, China; Beijing Key Laboratory for Helicobacter Pylori Infection and Upper Gastrointestinal Diseases (BZ0371), Beijing 100191, China.

Journal of genetics and genomics = Yi chuan xue bao
|April 4, 2024
PubMed
概括

缺少IL-17受体子单元CMTM4会通过改变肠道微生物组和增加S100a8/9.9来加剧性结肠炎 (UC). 恢复CMTM4功能可能会提供新的UC疗法.

关键词:
CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4 CMTM4我们的肠道微生物群.在IL-17受体C (IL-17RC) 中.在S100a8/9/9中,我们可以看到S100a8/9的情况.性结肠炎是一种

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