对低负荷血流限制运动和高负荷耐力运动的急性减痛,神经生理和感知反应
Ryan Norbury1, Ian Grant1, Alex Woodhead1
1Faculty of Sport, Technology and Health Sciences, St Mary's University, Twickenham, UK.
Experimental physiology
|April 5, 2024
概括
低负荷阻力运动与血液流量限制 (BFR) 增强疼痛缓解和减少神经抑制比高负荷运动更多. 与非BFR低负荷运动相比,BFR不会改变神经生理反应.
科学领域:
- 运动生理学 运动生理学
- 神经科学是一个神经科学.
- 康复 康复 康复 康复
背景情况:
- 抵抗运动对于康复至关重要,不同负载和血流限制 (BFR) 影响生理反应.
- 了解不同阻力运动方案的神经生理学和缺痛效应对于优化训练和恢复至关重要.
研究的目的:
- 为了比较低负荷耐力运动与BFR和没有BFR与高负荷运动的急性抗痛和神经生理效应.
- 为了研究疼痛压力值 (PPT),皮质脊髓刺激能力 (CSE) 和在这些运动条件下抑制的变化.
主要方法:
- 参与者接受了四种运动条件:低负荷 (LL),LL与BFR (BFR40,BFR80) 和高负荷 (HL).
- 神经生理学措施包括CSE,皮质脊髓抑制和通过跨磁刺激进行短间隔皮质内抑制 (SICI).
- 疼痛压力值 (PPT) 和最大自愿力 (MVF) 在多个时间点在运动前后被评估.
主要成果:
- 在不同条件下,没有观察到CSE或SICI的显著差异.
- 与高负荷运动相比,在所有低负荷条件 (包括BFR) 中,皮质脊髓抑制显著降低.
- 与LL相比,在BFR40,BFR80和HL条件下,PPT显著增加,表明更大的缺痛症.
结论:
- 低负荷的BFR阻力运动会引起更大的缺血,并比高负荷运动更有效地降低皮质脊髓抑制.
- 与非BFR低负荷运动相比,BFR不会改变急性神经生理反应.
- 低负荷BFR运动是一个可行的和有效的康复工具.
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